Basic Knowledge of ECG and Detailed Explanation of Common Clinical ECGs
Whether you are a healthcare professional, or just interested in learning more about ECGs, you will find this article extremely useful. It will provide you with a quick and basic overview of ECGs Medical record paper and a detailed explanation of some of the most common clinical ECGs.
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QRS complex
Using an ECG, you can identify problems with your heart. These include abnormalities in the heart rate, heart rhythm, and the ventricles. There are also symptoms such as lightheadedness, fatigue, chest discomfort, and palpitations.
A normal ECG will show predictable components. These include the P wave, the S wave, and the QRS complex. These waves are generated during depolarization of the ventricles. You can identify abnormalities if any of these waves are not present on the ECG paper.
The P wave is an impulse from the atria to the ventricles. The atria may be depolarized by atrial fibrillation, left atrial hypertrophy, or pulmonary hypertension. The action potential then passes through the AV node. The impulse may then spread to the ventricles. The ventricles may also be depolarized by spontaneous action potentials discharged within the ventricles.
The P wave is followed by a negative S wave. The S wave is the first downward deflection of the complex. The S wave may be followed by a T wave. The T wave represents the end of ventricular systole and repolarization. The T wave is longer than the QRS complex.
The QRS complex is composed of three closely related waves. The first wave is the R wave. The R wave is an upward deflection. The next downward deflection is the S wave. The QRS complex lasts from 80 to 100 milliseconds.
ST segment
During a cardiac cycle, the ST segment represents the plateau phase of the ventricular transmembrane action potential. It is also the period in which the muscular layer of the heart maintains contraction.
When a patient has an acute myocardial infarction, the ST segment is usually elevated. However, this can also represent a number of other causes. It is important to understand the different morphologic features of ST segment elevation and depression in order to properly diagnose the condition. In addition, this can help prevent an emergent coronary angiography.
There are several different ST segment morphologic features that can be recorded by different leads. This can help to differentiate the condition in a patient's ECG Medical recording paper. However, there are some conditions that are common and can mimic an acute myocardial infarction on electrocardiographic analysis.
Pericarditis is one example of a condition that causes a ST segment elevation on the electrocardiogram. This is the result of an injury current in the pericardial tissue. This current is probably smaller in pericarditis than in an acute myocardial infarction.
Another common condition that causes ST elevation is an electrolyte disturbance. A raised intracranial pressure can cause widespread deep T-wave inversions known as cerebral T waves. In addition, injury currents can lead to ST elevation. Using aspirin, nitrates, or morphine can help normalize electrocardiographic abnormalities.
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P wave
Detailed explanation of common clinical P wave is essential for an accurate interpretation of an ECG. ECG is an electrical signal that reflects repolarisation of myocardium. An ECG Medical Paper may have reproducible components and therefore can be used for clinical diagnosis.
The ECG is made up of a number of waves, each of which has a specific morphology and duration. The P wave is the first wave of electrical signal on an ECG. It is usually a smooth and rounded wave with a duration of 80 to 100 milliseconds. It originates from the atria and is followed by the tall QRS wave.
The P wave is usually biphasic, meaning that the amplitude is higher in lead II than lead V1. However, there is a wide range of normal P wave morphology and duration. In addition, there are pathological variants.
The P wave can also be notched. A notched P wave is a sign of left atrial hypertrophy. This is caused by increased resistance to empty blood into the right ventricle. This can be due to pulmonary artery pressure or tricuspid stenosis.
Lead V1 may also display a negative deflection. This type of deflection is often referred to as a U wave. The amplitude of a U wave is usually 1/3 of the amplitude of a T wave.
